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In nerves exposed to a hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose additional vitality substrate is required when the tissue power demand increases. Such data are essential as they show that glycogen has an vital position below physiological conditions. The truth that glycogen supported CAP conduction in the absence of exogenously applied glucose probably has no worth in ascribing a job to glycogen, since aglycaemia is an synthetic experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus can't be glycogen’s essential position. However supplying lactate to axons when tissue power demand increases implies a novel role for brain glycogen and suggests it acts as an energy buffer rather than an energy reserve, whose function is not to single-handedly support conduction, but to act in concert with ambient ranges of normoglycemic glucose.

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